Amplification of the chromosome segment 12q13–15, which carries the oncogenes MDM2, CDK4, and HMGA2, is found in WDLS and DDLS [3]; translocation of the genes FUS and DDIT3 (CHOP) genes are present in MLS [4], while loss of p53 and Rb oncosoppressors characterizes PLS and causes karyotype disorders [5]. This evidence concerns the gene FUS and McLeod neuroacanthocytosis syndrome.