This was elegantly demonstrated by Kudva et al., who showed that, in line with our results, infection with Staphylococcus aureus 6 days after influenza resulted in increased pulmonary levels of innate cytokines such as IL-6 and MCP-1, and increased neutrophil influx to the lungs, but decreased concentrations of T-cell-derived IL-17 and IL-22, which were demonstrated to play a pivotal role in fending off the staphylococcal infection [21]. The gene discussed is CCL2; the disease is infection.