Alzheimer’s disease (AD) is characterised pathologically by the abundant presence of deposits of amyloid β protein (Aβ) in the brain parenchyma in the form of amyloid plaques, and in blood vessel walls as cerebral amyloid angiopathy (CAA), and neurofibrillary tangles (NFT) composed of hyperphosphorylated tau proteins [19, 35]. This evidence concerns the gene MAPT and Alzheimer disease.