TGFB1 and pulmonary fibrosis: Considering the anti‐fibrotic effect of ASV in BLM‐induced pulmonary fibrosis, the possible mechanisms of that effect and the characteristics of EMT in pulmonary fibrosis, we hypothesized that ASV could suppress TGF‐β1‐induced phosphorylation of Akt and inactivity of FOXO3a and subsequently inhibit EMT.