TGFB1 and idiopathic pulmonary fibrosis: As a result of the notion that EMT directly contributes to myofibroblast accumulation in the lung,5, 29 groups of studies tried to find efficient approaches to inhibit or reverse this progress mediated by TGF‐β1.30, 31, 32 We for the first time identified the increased TGF‐β1 production was potently suppressed by ASV in BLM‐induced IPF.