We have confirmed the involvement of NKG2D on γδTc and MICA/B on MCF-7 and T47D in cytotoxicity of γδTc against breast tumor targets (Figure 4), although differences in the ability of γδTc to kill targets pre-incubated in hypoxia or normoxia do not appear to be related to surface levels of MICA (Figure 6). Here, KLRK1 is linked to breast neoplasm.