However, it is not easy to induce BEAS-2B cells to express eotaxin using TNF-α, and previous experiments showed that TNF/IL-4 could stimulate BEAS-2B cells to express eotaxin, which attracted eosinophils into allergic airways and lung tissue in asthma (Huang et al., 2014). The gene discussed is TNF; the disease is asthma.