SRF and deep vein thrombosis: MiR‐483‐5p is down‐regulated under hypoxia condition and can inhibit the growth of HUVECs by targeting serum response factor (SRF), which recedes wound healing and tube formation.110 Consistent with this, Kong et al111 found miR‐483‐3p can inhibit angiogenesis, up‐regulated in EPCs of DVT patients, which can inhibit EPC migration and tube formation and enhance apoptosis in vitro by targeting SRF, thus, reducing EPC homing and thrombus organization and recanalization in rat inferior vena cava thrombosis model.