Myasthenia gravis (MG) is a well-recognized antibody-mediated disease affecting the neuromuscular junction, caused by immunoglobulin G (IgG)1- and IgG3-complement, activating antibodies against the nicotinic acetylcholine receptor (AChR, AChR-Ab) in around 85% of patients [4]. This evidence concerns the gene IGHG3 and myasthenia gravis.