P-selectin captures leucocytes from the blood to bring them into contact with the endothelial cell surface on the blood vessel wall where P-selectin–PSGL-1 interaction supports leucocyte rolling, platelet activation, and aggregation, which leads to a cascade of reactions that promote inflammation and thrombosis; therefore, we can conclude that the platelet count is associated with the inflammatory reaction of Kawasaki disease and can speculate that the number of platelets is positively correlated with the severity of inflammation. The gene discussed is SELP; the disease is Kawasaki disease.