TP53 and infection: miR-125b seems to be associated with HPV-induced carcinogenesis in two distinct pathways: a) it has homology for HPV-L2, which is needed for the viral capsid assembly (so it is possible that after early infection miR-125b can inhibit HPV replication and improve viral clearance); and b) miR-125b leads to p53-pathway inactivation, thus maintaining cells viable with viral genomes inside.