EC tumors are associated with KRAS, beta-catenin (CTNNB) and PTEN mutations as well as microsatellite instability [5] Type II tumors, including high-grade serous carcinomas, are associated with frequent TP53 mutations and frequent copy number alterations in high-grade serous carcinomas [5, 27, 28]. The gene discussed is KRAS; the disease is serous adenocarcinoma.