A number of oncogenes, such as MYC, hypoxia inducible factors-1 alpha subunit (HIF1A), and tumor suppressor p53 (Yeung et al., 2008) as well as some pathways, such as phosphoinositide 3-kinase (PI3K/AKT) and protein kinase B (PKB), or the mammalian target of rapamycin (MTORC1) one (Wieman et al., 2007), are known to be involved in the energy metabolism regulation of cancer cells. This evidence concerns the gene AKT1 and cancer.