AKT1 and nasopharyngeal carcinoma: Similar to that, uc010rul‐siRNA‐transfected cells significantly inhibited the p‐PI3K, p‐Akt, and p‐mTOR (P < .01), but overexpressed ARHGAP42‐transfected reversed the effect incompletely (P < .01) in Figure 6F. Thus, we could imply ARHGAP42 promotes migration and invasion of nasopharyngeal carcinoma cells in vitro; the antisense lncRNA may be involved in this effect.