CCL7, which is irregularly released by immature monocytic myeloid cells (IMMCs) or neutrophils, somewhat increases the migration and invasion of human non-small cell lung cancer cells, suggesting the possibility that CCL7 exerts its carcinogenesis properties as a chemoattractant for neutrophils involved in the formation of the tumor microenvironment (Durrans et al., 2015; Michalec et al., 2002). The gene discussed is CCL7; the disease is non-small cell lung carcinoma.