These findings were confirmed by those recently published on the proteome profile changes after acute myocardial infarction (AMI) in an experimental model, where the expression levels of DBP and vitamin D receptor (VDR) were found increased in both left ventricular tissue of AMI mouse model, suggesting that DBP might be involved in left ventricular remodeling, and H9C2 cells, promoting cardiomyocyte apoptosis, after hypoxia [37]. This evidence concerns the gene VDR and myocardial infarction.