PPRC1 and Miyoshi myopathy: Therefore, one can assume that the functional neural mechanisms of pathological MM, mainly an overactivation of bilateral PrC46, might also be relatable to a structural level under healthy conditions in form of a gradual continuum from healthy to diseased state, reflecting a general inhibitory mechanism of inappropriate behavior (involuntary innervations of resting muscles) that is potentially mediated by transcallosal connections between bilateral PrC during unilateral contractions.