Therefore, the core level machinery regulating the crosstalk between autophagy and apoptosis that is targeted and thus highlighted by α-BSB-induced phenomena may lead on the one hand to a better understanding of the role of BCL2-family molecules in oncogenesis and tumor progression, while on the other hand it may represent a target for refined therapeutic tools, such as α-BSB itself and other agents that can affect the apoptosis/autophagy balance [22–24]. Here, BCL2 is linked to neoplasm.