TP53 and cervical carcinoma: B‐Myb is a transcription factor with specific DNA‐binding activity.36 Previous studies have shown that B‐Myb is overexpressed in cervical cancers.37 Importantly, the expression of B‐Myb mRNA peaks during the G1/S phase transition.38 In addition, overexpression of B‐Myb bypassed p53‐induced G1 arrest.39 Notably, B‐Myb was reported to bind to the Cdk1 promoter and activate the expression of Cdk1.23 It is well documented that B‐Myb was up‐regulated in HPV E7‐expressing cells.40 However, the expression and function of B‐Myb in 16E6‐expressing cells have not been reported.