In this way, 60 (5.5%) definite/probable FH, 362 (33.1%) possible FH and 671 (61.4%) unlikely FH were identified, suggesting 15.5% [(71–60)/71] of definite/probable FH might be explained by cholesterol from Lp(a) in this population (Supplemental Fig. 1). Here, LPA is linked to familial hyperaldosteronism.