SOD1 and epilepsy: The incremental SOD activity in the epileptic group subjects suggests efficient conversion of superoxide radicals to less toxic H2O2, and a reduction in the accumulation of superoxide radicals, and a compensatory mechanism reduced GPx activity or decreased the antioxidant activities of GPx and vitamin C and increased the activity of MDA, suggesting a reduced antioxidant defense capacity in epilepsy patients, and not one ameliorated by antiepileptic treatment.