Considering that many cells produce CCL2 including tumor cells, non‐malignant astrocytes, endothelial cells, and macrophages and microglia in a brain tissue context 46, how much of the CCL2 in glioblastoma may be attributed to Δ133p53β, and whether this contribution is sufficient to affect tumor progression, remains to be determined. This evidence concerns the gene CCL2 and neoplasm.