These experiments revealed that combined treatment of CNASB and IR similarly sensitized both, oxic and anoxia-tolerant NCI-H460 cancer cells, to the cytotoxic action of IR under Nx and hypoxic (Hx) conditions in short-term proliferation (Figure S4A in Supplementary Material) and long-term survival assays (Figure S4C in Supplementary Material) corroborating the radisensitizing action of BTA at the level of SLC25A1. The gene discussed is SLC25A1; the disease is cancer.