A characteristic morphological injury pattern in renal tissue, such as interstitial edema, renal tubular necrosis and inflammatory cell infiltration, and elevated AKI biomarkers such as neutrophil gelatinase-associated lipocalin (NGAL) and cystatin C were observed in a swine model of AKI after CA with ventricular fibrillation and asphyxiation [7]. The gene discussed is LCN2; the disease is ventricular fibrillation.