In LAR tumours, AR functionality is dependent on FOXA1: FOXA1 is required for AR binding chromatin, AR transcriptional activity and cell growth, directing AR to sites normally occupied by ER in luminal tumours, inducing an oestrogen-like programme stimulating proliferation.8 Our team has shown that AR + /FOXA1 + TNBC even tend to behave like luminal tumours.9 These two biomarkers could be useful to identify a particular subgroup of TNBC. This evidence concerns the gene AR and neoplasm.