ITGAM and stroke disorder: As the area of CD11b+ immunolabelling is increased after stroke (Figure 1B) and percentage of CD11b+SJC4+ co-localization remains unchanged, it can be concluded that the density of F4/80+CD11b+ monocyte-derived RPM does indeed increase after experimental stroke, and there is a proportional increase in CD11b+SJC4+ neutrophils also.