The EMT is a critical regulator of the CSC phenotype, and studies have reported that the EMT can generate tumor cells, and even normal cells, with CSC‐like properties.3, 27, 28 We had proved that FLI‐1 correlates positively with the EMT, and silencing FLI‐1 inhibited the expression of mesenchymal markers. This evidence concerns the gene FLI1 and neoplasm.