The cell population that characterizes chronic HCMV infection would show increased levels of NKG2C activating receptor, decreased levels of NKp30 and NKp46 receptors, increased levels of inhibitory receptors KIR and CD85j (ILT2/LIR-1), and decreased levels of the inhibitory receptor NKG2A (60, 63–66, 80). This evidence concerns the gene LILRB1 and cytomegalovirus infection.