Since inflammatory nature of atherosclerosis have been demonstrated both in animal models (Cybulsky and Gimbrone, 1991; Ross, 1999; Cybulsky et al., 2001) as well as in clinical studies (Schonbeck and Libby, 2004; Kinlay et al., 2008; Zhao, 2009) a link has been suggested between high sensitivity C-reactive protein (CRP) with coronary artery disease (CAD) risk (Ridker and Silvertown, 2008; Puri et al., 2013; Halcox et al., 2014; Quaglia et al., 2014). This evidence concerns the gene CRP and coronary artery disorder.