Intermittent aerobic exercise (80–90% maximum oxygen consumption rate) can significantly improve protein expression levels of PPAR-γ, PGC-1α, and mitochondrial transcription factor in cardiomyocytes postmyocardial infarction [88], illustrating that exercise training can improve mitochondrial biosynthesis postmyocardial infarction. Here, PPARGC1A is linked to infarction.