Choi et al. stated that ST inhibited 2,4-dinitrochlorobenzene- (DNCB-) induced atopic dermatitis in mice by the suppression of IgE, TNF-α, and IL-6 and by nuclear factor kappa-light-chain-enhancer of activated B cells (NF-κB) activation, as well as through mitogen-activated protein kinase (MAPK) activity [16]. Here, TNF is linked to atopic eczema.