Further, a few other studies have shown that protecting podocytes from hyperglycemia with a podocyte-specific deletion of the glucose transporter solute carrier family 2 or facilitated glucose transporter member 4 (SLC2A4, also known as GLUT4) [51] or from the resulting oxidative stress [12] can prevent diabetes-associated albuminuria without restoring normal levels of glucose. This evidence concerns the gene SLC2A4 and Hyperglycemia.