Furthermore, the decrease in lung eosinophilia of diabetic asthmatic animals can be explained by the lower levels of IL-5, CCL-11/eotaxin, and CCL-3/MIP-1α in its lungs, once IL-5 induces terminal maturation of eosinophils and prolongs eosinophil survival by delaying apoptosis death [40]; CCL-11/eotaxin-1 induces mobilization of eosinophils and their progenitors [41]; CCL-3/MIP-1α has chemotactic activity for eosinophils, although unlike CCL-11/eotaxin-1, it has no specific chemoattractant activity for eosinophils [42]. Here, IL5 is linked to Increased total eosinophil count.