Furthermore, a Malat1 knockout mouse model resulted in normal pre- and postnatal development and Malat1 inhibition in a mouse model for luminal B breast cancer gave rise to poorly developed metastatic tumors, suggesting that MALAT1 inhibition may be a feasible approach to reduce tumor growth and metastasis with minimal adverse effects on normal tissue [40, 41]. The gene discussed is MALAT1; the disease is breast carcinoma.