ANXA5 and hepatocellular carcinoma: Greater extent suppressions of these molecules were detected in Hca-P-ANXA5-shRNA1 than Hca-P-ANXA5-shRNA2, which consequently contributed in the greater extent suppressions of malignant phenotypes and behaviors of hepatocarcinoma cells, which also implicated the linkage of ANXA5 overexpression positively correlated with RAC1 and CRKI/II in promoting the clinical progression and metastasis of HCC.