To confirm these results, we took advantage of two mouse tumourgraft models carried out with ADC (UN-ADC12) or SQLC (UN-SCC680) cells derived from chemically-induced tumours and having received or not sunitinib, a VEGFR TKI, or DC101, a murine anti-VEGFR2 antibody.2 Consistent with our results in cell lines, increased sVEGFR1-i13 immunostaining was detected in UN-SCC680 tumourgrafts treated with both anti-angiogenic therapies compared to untreated tumourgrafts (Fig. 2a). Here, KDR is linked to neoplasm.