We further identified that the level of p-STAT3 was significantly decreased after the administration of iPSC-MSCs (n = 6) but not DEX (n = 6) (Fig. 4b), suggesting that iPSC-MSCs may inhibit the differentiation of Th17 cells in this model of neutrophilic airway inflammation via downregulating p-STAT3 level. The gene discussed is STAT3; the disease is inflammatory response.