Both RAD51 and BRCA2 were also high in the majority of IPF fibroblasts at similar time points to increased FoxM1 expression, further supporting that FoxM1 protects IPF fibroblasts from radiation-induced DNA damage via expression of RAD51 and BRCA2 (Fig. 3a, lower). Here, RAD51 is linked to idiopathic pulmonary fibrosis.