LGALS3 and lung adenocarcinoma: Similar as LPS, Galectin-3 overexpression also caused a dramatical increase in TLR4, MyD88 and p-p65 proteins and NEAT1 mRNA expression, which could also be partially reversed by CLI-095; in addition, the promotive effects of Galectin-3 on lung adenocarcinoma cell proliferation and migration was also partially attenuated by CLI-095.