However, infected Ccr2-/- mice, in which CD11b+Ly6Chigh monocytes recruitment to the infected tongue was strongly impaired (S9C and S9D Fig), displayed no defect in IL-17A production on day 1 post-infection by any of the three CD90+ cell subsets (TCRβ+, TCRγδ+, ILCs) compared to WT control mice (S9E Fig). Here, IL17A is linked to infection.