The influence of different: (i) anthracycline drugs, (ii) linkers among the tumor-homing peptide moiety and the drug, and (iii) tumor-homing peptides (e.g., GnRH-III and D-Lys6-GnRH-I) was examined regarding their in vitro cellular uptake, drug release and cytostatic effect [145]. The gene discussed is GNRH1; the disease is neoplasm.