NFKB1 and bronchopulmonary dysplasia: The in vitro recapitulation of the inflammatory milieu confirmed the dependency of phenotype alteration on NFκB. The classical proinflammatory cytokines including IL-1β, IL-6, IL-8, or TNF-α induced the identical changes as observed in MSCs from preterm infants with severe BPD, and NFκB targeting reverted the proinflammatory phenotype [81].