To examine whether the observed effect of MUC1 inhibition on susceptibility of AML cells to cytotoxic injury is conveyed via survivin‐mediated mechanism, survivin was overexpressed in MOLM‐14/CRISPR AML cells using lentiviral transduction (MOLM‐14/CRISPR/Survivin). This evidence concerns the gene MUC1 and acute myeloid leukemia.