Specifically, we examined the effect of MUC1‐C silencing on the expression of survivin, a target gene of the WNT/β‐catenin/TCF pathway, which is critical for leukaemia survival, resistance to apoptosis and disease progression.14, 16, 17, 21, 22, 23, 24, 25, 26, 27, 28, 29, 30. The gene discussed is HNF4A; the disease is leukemia.