NCR3 and HIV-1 infection: On the other hand, we found that NKp30 can also play an important role in HIV-1 infection upon its induced expression in Vδ1+ T cells; NKp30 ligation triggered the production of CCL3, CCL4, and CCL5 chemokines that suppressed the replication of a CCR5 tropic strain of HIV-1 (40).