The pro-tumoral function of γδ17 T cells was shown to result from either direct support of tumor cell survival, through the interleukin 6 (IL-6)–signal transducer and activator of transcription 3 (STAT3) axis [7,17], or indirect establishment of a prosperous environment for the tumor, especially through promotion of angiogenesis [8,15]. This evidence concerns the gene IL6 and neoplasm.