Several researchers found that PCs contributed to the prevention of UV-induced skin disorders, diabetic retina injury, or zearalenone-induced liver damage by activating Nrf2 pathway or inhibiting MAPK/NF-κB pathway, by scavenging hydroxyl radicals and superoxide anions, and by upregulating endogenous antioxidants and detoxication enzymes, such as hemeoxygenase-1 (HO-1), catalase (CAT), superoxide dismutase (SOD), and glutathione peroxidase (GSH-Px) [39–42]. This evidence concerns the gene HMOX1 and skin disorder.