There was a statistically significant interaction effect between stressor exposure and infection exposure on GPR41 expression (Fig 2E, p<0.05), but post-hoc testing indicated that this was due to significant increases in GPR41 in HCC and RST mice (p<0.05), but not FWD mice, when challenged with C. rodentium. Neither stressor exposure nor infection exposure affected GPR43 expression (Fig 2F) as indicated by the lack of significant main effects or an interaction effect in the two factor ANOVA. This evidence concerns the gene FFAR3 and hepatocellular carcinoma.