To address the essential partner function of HMGA2 in recruiting GCN5 to the MMP2 promoters in GBM cells, we performed ChIP experiments targeting HMGA2 and GCN5 in the stable U87MG and U251 cell lines with shRNA‐mediated knockdown of endogenous HMGA2 (U87MG‐HMGA2‐sh‐1, ‐sh‐2, U251‐HMGA2‐sh‐1, ‐sh‐2) and U87MG‐Scramble and U251‐Scramble cells. The gene discussed is MMP2; the disease is glioblastoma.