Furthermore, Snail1 arrests the epithelial cell cycle and promotes survival, by repressing the expression of pro‐apoptotic genes (Vega et al., 2004), whereas transcriptional repression of the fructose‐1,6‐bisphosphatase (FBP1) gene switches on glycolytic rates and contributes to the survival of breast cancer cells that undergo the EMT (Dong et al., 2013b), thus linking EMT to the generation of stem‐like features in cancer cells (Ye and Weinberg, 2015). The gene discussed is SNAI1; the disease is breast carcinoma.