Additionally, the results indicate that etomoxir-dependent inhibition of mitochondrial beta-oxidation in the EAE rat model is effective because of the regulation of serum autoantibodies against proteins such as ApoE, clusterin, SAP, and serum albumin, suggesting a role of these antigens in the progression of EAE and their potential use as diagnostic biomarkers for MS. This evidence concerns the gene ALB and myeloid sarcoma.