It has been shown that one mutation designated p.Cys681X in the LDLR gene accounts for FH in 81.5% of Lebanese FH patients [16] and an “Iceland-specific” splice-site mutation (c.698 + 2T4C [IVS4 + 2T4C]) in the LDLR gene was identified in 60% of the patients with FH in Iceland [17, 18]. Here, LDLR is linked to familial hyperaldosteronism.